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Proteomic Analysis of Kir6.2 Using Bioinformatics Software Tools

 

Marketed By :  LAP LAMBERT Academic Publishing   Sold By :  Kamal Books International  
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  • Product Description
 

KATP Channels couple glucose metabolism to membrane potential and thus play a central role in glucose stimulated insulin secretion. Genetic mutations in the genes encoding the channel subunits (Kir 6.2) can cause Congenital Hyperinsulinism (CHI). BLAST and FASTA similarity searches reveals the closely relatedness of Kir6.2 with other inwardly rectifying K+ channel family of proteins. The phylogenetic analysis showed the evolutionary relationship of various mammals with reference to Kir6.2. The secondary structure prediction, Tranmembrane and hydropathy prediction reveals the Kir6.2 contained more number of helical regions.Further, the Kir6.2 protein contains motifs which are responsible for Prokaryotic membrane lipoprotein lipid attachment site, cAMP and cGMP dependent protein kinase, casein kinase II and Protein Kinase C phosphorylation site.

Product Specifications
SKU :COC54125
AuthorPaulraj Sugapriya Menaga
LanguageEnglish
BindingPaperback
Number of Pages104
Publishing Year2014-01-29T00:00:00.000
ISBN9783659513855
Edition1 st
Book TypeBiology, life sciences
Country of ManufactureIndia
Product BrandLAP LAMBERT Academic Publishing
Product Packaging InfoBox
In The Box1 Piece
Product First Available On ClickOnCare.com2015-06-08 00:00:00
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